Two photon-linked biodiversity (2)

By: James V. Kohl | Published on: December 29, 2018

Unravelling mystery of how, when DNA replicates

…they found that these three elements together were the key to DNA replication.

“Removing these elements shifted the segment’s replication time from the very beginning to the very end of the process,” Gilbert said.

DNA replication is energy-dependent and RNA-mediated in the context of biophysically constrained pheromone-controlled viral latency (aka autophagy), which prevents nearly all the transgenerational epigenetic inheritance of the virus-driven degradation of messenger RNA, which has consistently been linked from mutations to all pathology.
Who, besides Jay R. Feierman, is claiming that the mysteries of DNA replication have not been unraveled:

Variation is not nutrient availability and the something that is doing the selecting is not the individual organism. A feature of an educated person is to realize what they do not know. Sadly, you don’t know that you have an incorrect understanding [of] Darwinian biological evolution.

See also:

I am absolutely certain that if you showed this statement to any professor of biology or genetics in any accredited university anywhere in the world that 100% of them would say that “Random mutations are the substrate upon which directional natural selection acts” is a correct and true statement.

The final solution??? from 6/14/14

[MODERATOR NOTE: I’m not going to post more from Kohl until he answers the very direct and simple question posed to him by anon, which is whether he (Kohl) believes that RNA splicing can change DNA.]

I explained RNA splicing to Jay R. Feierman in 1995 at at presentation in Minot ND, that followed from book publication of “The scent of eros: mysteries of odor in human sexuality” (by Kohl and Francoeur 1995)
See also the molecular epigenetics section of our review:
From Fertilization to Adult Sexual Behavior (1996)

Small intranuclear proteins also participate in generating alternative splicing techniques of pre-mRNA and, by this mechanism, contribute to sexual differentiation in at least two species, Drosophila melanogaster and Caenorhabditis elegans (Adler and Hajduk, 1994; de Bono, Zarkower, and Hodgkin, 1995; Ge, Zuo, and Manley, 1991; Green, 1991; Parkhurst and Meneely, 1994; Wilkins, 1995; Wolfner, 1988). That similar proteins perform functions in humans suggests the possibility that some human sex differences may arise from alternative splicings of otherwise identical genes.

There are still very few people who support our truthful representation of how alternative splicings of microRNAs (formerly pre-mRNAs) are linked to the sexual differentiation of cell types in species from yeasts to humans. Most people realize that acceptance of the facts refutes their ridiculous theories about sexual orientation.
For comparison of our facts to the pseudoscientific nonsense touted by theorists, see what’s next: ETHOLOGY, PSYCHOLOGY, PSYCHIATRY: AN EVOLUTIONARY APPROACH

The overall purpose of the Workshop is to discuss the implications of ethology and evolutionary psychology for psychological and psychiatric research and practice. It will focus on a diverse array of topics, including the analysis of nonverbal behaviour, behavioural ecology, particularly in the form of life history theory, and evolutionary genetics of psychiatric disorders. The format will involve talks by international authorities who have been engaged in such research. The workshop will be highly interdisciplinary including aspects involving behavioural and social neuroscience as well as psychopharmacology and psychotherapy

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